Reversing Alzheimer’s, Cognitive Decline, & Neurodegenerative Disease

It was good to hear and she had improvements and you know, as metabolism progresses, so does cognition, so you want to optimize all the trophic support, which is what we did for her. Be careful, obviously the very controversial area is bioidentical hormone replacement therapy and who here does BHRT? Some people again consider optimal

cognitive

function for someone who has

cognitive

impairment to include optimizing their hormones. For these people especially, a whole coffee fruit extract can be particularly helpful and we generally use a hundred milligrams once or twice a day because this increases BDNF and more and more work is being done on this.

Be careful with fasting, especially with people who have a BMI below 20, so we generally want to cycle them so that they do their fast for three or four days and then leave it for one day, not to say that they take a lot of sugar , but other things. like sweet potatoes and things include a little more complex carbohydrates in your diet so you don't lose too much weight and we mentioned synaptogenesis with citicoline and DHA. Is there a role for secretogogues? I'm particularly interested in that. Theoretically, after you start to improve, growth hormone should be helpful for these people and there have been trials in the past using it again as a monotherapy, so it's not saying much, they didn't work, but again it was a monotherapy. .

In theory you should be able to do it the way you are now, since you now have the right environment you should be able to make these people better, so we'll see over time if that works or not. So here's a guy with type 1.5, fasting insulin 14, hs-CRP, so you can see he doesn't have a lot of inflammation there, but he has insulin resistance, BMI was 33, copper, zinc, magnesium, etc. normal, so write. 1.5 and so you want to restore insulin sensitivity in these people and again, I think you have all of this in a pamphlet so I won't talk too much about this.

And then to finish, here is a woman with type 3, so she is a person exposed to toxins. This woman had a positive PET for amyloid, positive APOE4, her MoCA score was 24 out of 30, so she has mild cognitive impairment. The beginning was with depression and when someone arrives with depression, be careful, many times they are exposed to toxins. Non-amnestic presentation is another characteristic of type 3, and these people often begin their symptoms in their 50s, which also happened to this woman. They often have low zinc levels, low triglycerides, and a high copper:zinc ratio. She had all that.

Anti-amyloid therapy failed and actually made it worse, as we have seen. His C4a was high, his TGF-beta 1, he was MARCoNS positive, so he's been on the protocol for over a year and now his MoCA is back to a perfect 30, same as President Trump's, so we're happy Whether that could tell you that perhaps it is not a sensitive enough test, I don't know, but in any case I didn't say that, the neuropsychologist told me that. Very good, marked subjective improvement and you have been doing very, very well, your C4a has come down to normal, so in closing, what we would like to do now is ask if we can take the same approach and we can make the appropriate modifications to shine. in other

neurodegenerative

disease

s.

If we understand the biochemistry and the genetics and the epidemiology of each of them, can we really get this kind of approach where we look at multiple variables to work with these other ones and the ones that are underlined that we just started to look at? I saw the first people and I had some suggestions of success, so we are excited about the fact that this is where 21st century medicine is headed and we call it the Ark project because the ark, of course, was two by two and we want have it Some people with each of these, just to see if we look at these larger data sets, we can really have an impact on these people, so I'll end there so you know time's up.

Thanks a lot again for everything. you